What are 3 common complications of a myocardial infarction?

Complications of MI include arrhythmic, mechanical, and inflammatory (early pericarditis and post-MI syndrome) sequelae, as well as left ventricular mural thrombus (LVMT) (see the following image).

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One may also ask, can we give fluids in inferior wall MI?

Contrary to the practice of limiting fluids in MI patients without right ventricular involvement, fluids should be given to the RVI patient to maintain cardiac output, blood pressures and coronary artery filling pressures. Normal saline up to 40 ml/kg is the preferred fluid.

Besides, how do you treat inferior myocardial infarction? While heart blocks are a main contributor to morbidity and mortality, most high-degree heart blocks are treatable with atropine. It is seldom necessary to use a temporary pacemaker. The damaged myocardium can lead to potentially lethal arrhythmias such as ventricular tachycardia and ventricular fibrillation.

Regarding this, what are the potential complications that could occur post MI?

CONTENTS

  • Approach to the deteriorating post-MI patient.
  • Retroperitoneal hematoma.
  • Post-MI pericarditis.
  • Re-infarction.
  • Mechanical complications. Mitral valve chordae tendinae rupture. Ventricular septal defect (VSD) …
  • Tachyarrhythmia. Atrial fibrillation or atrial flutter. …
  • Bradyarrhythmia. Heart block s/p MI.

What does inferior MI affect?

Inferior wall myocardial infarction (MI) occurs from a coronary artery occlusion with resultant decreased perfusion to that region of the myocardium. Unless there is timely treatment, this results in myocardial ischemia followed by infarction.

What is the earliest indicator of inferior wall myocardial infarction MI?

In the earliest hours of acute IMI, the ST-segments in II, III and aVF may be normal or near-normal, but frequently, there is ST-segment depression in aVL. Thus, ST-segment depression in AVL constitutes a critical “early warning” sign of acute inferior wall STEMI.

What is the most common early complication of MI?

Pericarditis

  • Pericarditis is most common following an anterior infarction. …
  • The frequency is reduced with early reperfusion in the acute management of infarction.
  • Frequently occurs within a few days of the myocardial infarction and presents with a low-grade fever, pericardial friction rub and pleuritic chest pain.

Which heart block is associated with inferior wall MI?

Atrioventricular block in acute inferior wall myocardial infarction: harbinger of associated obstruction of the left anterior descending coronary artery.

Which leads are affected in inferior MI?

Inferior STEMI is usually caused by occlusion of the right coronary artery, or less commonly the left circumflex artery, causing infarction of the inferior wall of the heart [6, 7]. Upon ECG analysis, inferior STEMI displays ST-elevation in leads II, III, and aVF.

Why do you not give Nitro to inferior MI?

Nitroglycerin is also contraindicated in the setting of an inferior MI with right ventricular involvement because, in this specific situation, the heart is dependent on preload.

Why does inferior wall MI cause bradycardia?

Also, because the right coronary artery perfuses the sinoatrial node, heart block and bradycardia may occur. A high degree heart block, defined as a second or third-degree block, is seen in 19% of patients with acute inferior wall MI. The amount of collateral circulation to the AV impacts the rate of heart blocks.

Why does inferior wall MI cause left axis deviation?

Left axis deviation is seen in inferior MI VTs when the exit site is near the septum. As the VT exit moves from the midline toward the lateral (i.e., posterior) wall, the QRS axis becomes directed more rightward or superior.

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